@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix proteinModification: . @prefix psimod: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:97874; a Protein: . sub:_2 belv:variantOf mgi:101898; a proteinModification:, psimod:00696 . sub:_3 occursIn: species:10090; rdf:object sub:_2; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(MGI:Rb1) -> p(MGI:Pou2f1,pmod(P))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "reconstitution of Rb-defective tumor cells with functional Rb results in hyperphosphorylation of Oct1 and disruption of Oct1 interaction with H2A octamer element Oct1 can be posttranslationally modified by phosphorylation, and this modification has been shown to reduce the binding of Oct1 to DNA"; prov:wasQuotedFrom pubmed:11533238 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:11533238; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:31:43.369+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }