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http://www.tkuhn.ch/bel2nanopub/RAWRdgFRk0OWd19qMA18eANmxLael79NhmODQQGuaDeto#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RAWRdgFRk0OWd19qMA18eANmxLael79NhmODQQGuaDeto#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0016301
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http://purl.obolibrary.org/obo/RO_0002204
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kin(p(RGD:Akt1)) => p(RGD:Bad,pmod(P,S,136))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Our data demonstrated that Akt1 mediated the phosphorylation of Bad at serine 136, which increased the interaction of serine 136-phosphorylated Bad with 14-3-3 proteins and prevented the dimerization of Bad with Bcl-Xl, inhibited the release of cytochrome c to the cytosol and the death effector caspase-3 activation, leading to the survival of neuron.
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Selventa
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2014-07-03T14:33:10.895+02:00
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