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> .
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@prefix ncit: <
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> .
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http://linkedlifedata.com/resource/umls/id/
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http://identifiers.org/ncbigene/
> .
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http://identifiers.org/pubmed/
> .
@prefix eco: <
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> .
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> .
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http://www.w3.org/ns/prov#
> .
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http://purl.org/pav/
> .
@prefix prv: <
http://purl.org/net/provenance/ns#
> .
@prefix dcterms: <
http://purl.org/dc/terms/
> .
@prefix np: <
http://www.nanopub.org/nschema#
> .
@prefix dgn-np: <
http://rdf.disgenet.org/resource/nanopub/
> .
@prefix dgn-gda: <
http://rdf.disgenet.org/resource/gda/
> .
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http://rdf.disgenet.org/v4.0.0/void/
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ncit:C16612
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a
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dgn-np:NP259892.RAWAnfwz2rUaf1csyS_J74kgIfxMZtEDbVLT9z4kZRYzA130_assertion
dcterms:description
"[Using this approach, we also identified the other major Glut1 3'UTR RNA binding activity as hnRNP L. Stimuli (hypoxia and hypoglycemia) which increase Glut1 mRNA stability selectively decreased polysomal levels of hnRNP A2 and L. Immunoprecipitation demonstrated that hnRNP A2 and L exist as a complex in vivo.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
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xsd:date
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a
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xsd:dateTime
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