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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAVu99X2dyWDzSqqzEsqeNKQDQkRm7zRxhkKKL0tfBS7Q#_4 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAVu99X2dyWDzSqqzEsqeNKQDQkRm7zRxhkKKL0tfBS7Q#_3 http://www.w3.org/ns/prov#value In platelets, glucose uptake can be stimulated by thrombin or insulin and is primarily mediated by the glucose transporter, GLUT3 [40]. Glucose uptake stimulated by thrombin or insulin is reduced in the presence of the Akt inhibitor, ML-9, suggesting that Akt contributes to glucose uptake in platelets, as it does in skeletal muscle or adipocytes [40]. 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