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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAVtwEIlUBzaSGDwbi4_gN5NGJ6hyASTlIfW0DyIcfHWY#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=6018
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://purl.obolibrary.org/obo/BFO_0000066
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a(SCHEM:Dinoprostone) -| (p(HGNC:IL6) -> p(HGNC:STAT3,pmod(P)))
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p(HGNC:IL6) -> p(HGNC:STAT3,pmod(P))
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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The effects of IL-6 in ECs are mediated by interaction of sIL-6Ralpha/IL-6 complexes with gp130 dimers at the cell surface (30)......Importantly, similar inhibitory effects on STAT3 phosphorylation in response to sIL-6Ralpha/IL-6 were observed upon activation of endogenous G protein-coupled receptors for PGE2 (via EP2 and EP4 receptors), CGS21680 (A2A adenosine receptor [A2AAR]) and alphaMSH (melanocortin receptor 1), each of which are known to activate adenylyl cyclase and elevate cAMP (11, 14, 33, 44) (Fig. 1B).
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http://www.w3.org/ns/prov#wasQuotedFrom
http://www.ncbi.nlm.nih.gov/pubmed/16914720
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Selventa
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http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RAVtwEIlUBzaSGDwbi4_gN5NGJ6hyASTlIfW0DyIcfHWY#pubinfo
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2014-07-03T14:33:02.739+02:00
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