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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAVUGst9u1uh5Zzw7tjDvbvKdj-K7-C9cI04uKbzBSF_I#_7 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAVUGst9u1uh5Zzw7tjDvbvKdj-K7-C9cI04uKbzBSF_I#_6 http://www.w3.org/ns/prov#value PI3K-Akt-GSK3b pathway Glycogen-synthase kinase 3b (GSK3b) is a distinct substrate of Akt1 that has been shown to modulate hypertrophy. GSK3b activity is inhibited by Akt1 phosphorylation [65] (Fig. 1). Expression of a dominantnegative, kinase-inactive form of GSK3b induces dramatic hypertrophy in skeletal myotubes [6]. 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