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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
http://www.tkuhn.ch/bel2nanopub/RAVB6O69aEYyfrInlrdKQ_BENQYRQ_GDbE2R28wbQYQUQ#assertion
http://resource.belframework.org/belframework/20131211/namespace/selventa-named-complexes/Nfkb%20Complex
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0043234
http://www.tkuhn.ch/bel2nanopub/RAVB6O69aEYyfrInlrdKQ_BENQYRQ_GDbE2R28wbQYQUQ#_1
http://semanticscience.org/resource/SIO_000139
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http://amigo.geneontology.org/amigo/term/GO:0042789
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=5344
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tscript(complex(SCOMP:"Nfkb Complex")) -> r(HGNC:ICAM1)
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http://purl.org/dc/elements/1.1/description
Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Cytokines (e.g., TNF-alpha and several interleukins) and constituents of modified lipoproteins (e.g., lysophosphatidyl choline) enhance oxidative stress in endothelial cells, which in turn activates the NFKB signaling pathway. Activated NFkB migrates into the nucleus and induces the expression of numerous genes, including many pro-inflammatory genes such as P-Selectin, ICAM-1 and VCAM-1.
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Selventa
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2014-07-03T14:33:40.148+02:00
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