@prefix this: <http://rdf.disgenet.org/nanopublications.trig#NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix sio: <http://semanticscience.org/resource/> .
@prefix ncit: <http://ncicb.nci.nih.gov/xml/owl/EVS/Thesaurus.owl#> .
@prefix lld: <http://linkedlifedata.com/resource/umls/id/> .
@prefix miriam-gene: <http://identifiers.org/ncbigene/> .
@prefix miriam-pubmed: <http://identifiers.org/pubmed/> .
@prefix eco: <http://purl.obolibrary.org/obo/eco.owl#> .
@prefix wi: <http://purl.org/ontology/wi/core#> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix pav: <http://purl.org/pav/2.0/> .
@prefix prv: <http://purl.org/net/provenance/ns#> .
@prefix dcterms: <http://purl.org/dc/terms/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix dgn-np: <http://rdf.disgenet.org/nanopublications.trig#> .
@prefix dgn-gda: <http://rdf.disgenet.org/gene-disease-association.ttl#> .
@prefix dgn-void: <http://rdf.disgenet.org/v2.1.0/void.ttl#> .
dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_head {
  this: np:hasAssertion dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_assertion ;
    np:hasProvenance dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_provenance ;
    np:hasPublicationInfo dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_publicationInfo ;
    a np:Nanopublication .
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  dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_provenance a np:Provenance .
  dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_publicationInfo a np:PublicationInfo .
}
dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_assertion {
  miriam-gene:3320 a ncit:C16612 .
  lld:C3469521 a ncit:C7057 .
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    a sio:SIO_001121 .
}
dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_provenance {
  dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_assertion dcterms:description "[In conclusion, our results identify FANCA as a novel client of Hsp90, suggesting that Hsp90 promotes activation of the FA pathway through regulation of intracellular turnover and trafficking of FANCA, which is critical for cellular tolerance against genotoxic stress.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
    wi:evidence dgn-void:source_evidence_literature ;
    sio:SIO_000772 miriam-pubmed:17327415 ;
    prov:wasDerivedFrom dgn-void:befree-20140225 ;
    prov:wasGeneratedBy eco:ECO_0000203 .
  dgn-void:befree-20140225 pav:importedOn "2014-02-25"^^xsd:date .
  dgn-void:source_evidence_literature a eco:ECO_0000212 ;
    rdfs:comment "Gene-disease associations inferred from text-mining the literature."@en ;
    rdfs:label "DisGeNET evidence - LITERATURE"@en .
}
dgn-np:NP189050.RAVAk6QeJgg9wfBJUQSeB7lPsooeNPOMcy4FL19Ybski8130_publicationInfo {
  this: dcterms:created "2014-10-02T12:33:44+02:00"^^xsd:dateTime ;
    dcterms:rights <http://opendatacommons.org/licenses/odbl/1.0/> ;
    dcterms:rightsHolder dgn-void:IBIGroup ;
    dcterms:subject sio:SIO_000983 ;
    prv:usedData dgn-void:disgenetrdf ;
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}