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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAUbbm9cvioKGZJQ9FKfwaPsUyFX0FoSE9bGmMkZmg9NM#_5 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAUbbm9cvioKGZJQ9FKfwaPsUyFX0FoSE9bGmMkZmg9NM#_4 http://www.w3.org/ns/prov#value Inhibition of the RhoA/ROK pathway is perhaps the most important mechanism of Ca2 desensitization by cGMP, as first shown by the dramatic reversal of GTPS (and agonist)-induced Ca2 sensitization of force and RLC phosphorylation in smooth muscle (176, 429) and recently by the effect of upstream stimulation of PKG by NO (65). (65). Chitaley K and Webb RC. Nitric oxide induces dilation of rat aorta via inhibition of rho-kinase signaling. 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