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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAU061mxlCNuu_hPfgl0pOTXTQL1K394hH3Y3s3Po8KVc#_6 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAU061mxlCNuu_hPfgl0pOTXTQL1K394hH3Y3s3Po8KVc#_5 http://www.w3.org/ns/prov#value PU.1 and Spi-B directly regulate the expression of several genes associated with BCR signaling such as BLNK, B220, Btk, P2Y10, and Grap2 [10, 29–32].Spi-C has DNA-binding specificity identical to PU.1 and Spi-B and therefore might regulate many of the same genes as these transcription factors [25].The levels of BLNK, Lyn, B220, Btk, and P2Y10 transcripts were significantly decreased in bone marrow pre-B and immature B cells enriched from Spi-C transgenic mice compared with the wild type. 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