@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix schem: . @prefix go: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: obo:CL_0000233, obo:EFO_0002795, species:9606; rdf:object go:0006979; rdf:predicate belv:increases; rdf:subject schem:Advanced%20Glycation%20End%20Product; a rdf:Statement . sub:assertion rdfs:label "a(SCHEM:\"Advanced Glycation End Product\") -> bp(GOBP:\"response to oxidative stress\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "20131211" . sub:_2 prov:value "Since AGEs elicited superoxide generation in platelets and that superoxide dismutase or indomethacin abolished the enhancing effects of AGEs on the platelet aggregation, AGEs could augment the platelet aggregation through oxidative stress generation and subsequent prostanoid production (Hangashi et al., 1998)."; prov:wasQuotedFrom pubmed:16472128 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:16472128; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dct:created "2014-07-03T14:32:56.156+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }