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http://www.tkuhn.ch/bel2nanopub/RASVCQaYB9Fw_niqbHksHo8En9xjXpIPz21TUu4Dgz6Vg#_1
http://semanticscience.org/resource/SIO_000139
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://purl.obolibrary.org/obo/RO_0002204
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http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=7797
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.w3.org/1999/02/22-rdf-syntax-ns#Statement
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http://purl.obolibrary.org/obo/BFO_0000066
http://purl.obolibrary.org/obo/CL_0000235
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http://purl.obolibrary.org/obo/BFO_0000066
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a(CHEBI:"sphingosine 1-phosphate") -| (cat(p(HGNC:TLR2)) -| p(HGNC:NFKBIA))
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cat(p(HGNC:TLR2)) -| p(HGNC:NFKBIA)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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The activation of TLR2 by Pam3CSK4 induced the degradation of the NF-kB inhibitor IkBa in human monocyte-derived macrophages. NF-kB activation was blocked by S1P at low micromolar concentrations. Statistically significant inhibition of TLR2-dependent NF-kB activation was observed with 1 mM S1P, which argues in favour of a receptor-dependent and physiologically relevant effect (Figure 4A).
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.w3.org/ns/prov#wasDerivedFrom
http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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http://www.tkuhn.ch/bel2nanopub/RASVCQaYB9Fw_niqbHksHo8En9xjXpIPz21TUu4Dgz6Vg#_6
http://www.tkuhn.ch/bel2nanopub/RASVCQaYB9Fw_niqbHksHo8En9xjXpIPz21TUu4Dgz6Vg#pubinfo
http://www.tkuhn.ch/bel2nanopub/RASVCQaYB9Fw_niqbHksHo8En9xjXpIPz21TUu4Dgz6Vg
http://purl.org/dc/terms/created
2014-07-03T14:33:19.996+02:00
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http://orcid.org/0000-0001-6818-334X
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