@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix RNA: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:1201673; a Protein: . sub:_2 geneProductOf: mgi:99829; a RNA: . sub:_3 occursIn: obo:UBERON_0002481, species:10090; rdf:object sub:_2; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(MGI:Shox2) -> r(MGI:Runx2)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "We flanked the entire coding region of Shox2 with LoxP sites (10), such as to induce deletion upon exposure to the Cre recombinase (Fig. 1). Mice carrying such a floxed allele as well as a fully deleted copy and the limb-specific Prx1-Cre transgene (11) (hereafter referred to as Shox2 c/?) were thus devoid of any Shox2 transcript in their developing limbs (Fig. 1 b and c). We looked at Runx2 (mRNA) expression in Shox2 c/- mice, and, strikingly, very few Runx2-positive cells were scored in the humerus at E12.5 (Fig. 5 a)."; prov:wasQuotedFrom pubmed:16537395 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:16537395; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:32:57.116+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }