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http://www.tkuhn.ch/bel2nanopub/RAR0KgTgFeZe3NV1sE3ljd4JPpOq0rVVUsldSO_mCF7yQ#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=3229
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http://semanticscience.org/resource/SIO_000139
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http://amigo.geneontology.org/amigo/term/GO:0016301
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http://purl.obolibrary.org/obo/RO_0002204
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http://purl.obolibrary.org/obo/BFO_0000066
http://disease-ontology.org/term/DOID:1107
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http://purl.obolibrary.org/obo/BFO_0000066
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http://www.w3.org/2000/01/rdf-schema#label
p(HGNC:EGF) -> kin(p(HGNC:AKT1))
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Western blotting with the anti-phospho-Akt1 antibody, which recognizes only phosphorylated Ser-473 residue, was performed in human esophageal cancer cell lines (TE-2, TE-5, TE-8, TE-9, TE-10, and TE-12 cells) (top panel). PDGF-treated NIH3T3 cells served as a positive control. The phospho-Akt antibody demonstrated two bands at 60 kDa. The phosphorylation of Akt was enhanced by EGF stimulation in all the esophageal cancer cell lines examined, although TE-5 cells showed high basal phosphorylation of Akt.
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Selventa
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http://purl.org/dc/terms/created
2014-07-03T14:31:36.333+02:00
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