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http://www.tkuhn.ch/bel2nanopub/RAQe4KjBP8ji59oLd2QgL-l8swImbwD_rl16LamosOjZk#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=10972
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http://amigo.geneontology.org/amigo/term/GO:0006306
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bp(GOBP:"DNA methylation") -| r(HGNC:SLC22A8)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Electrophoretic mobility shift assays demonstrated binding of the HNF1alpha/HNF1alpha homodimer or HNF1alpha/HNF1beta heterodimer to the hOAT3 promoter. It was also demonstrated that the promoter activity of hOAT3 is repressed by DNA methylation. Moreover, the expression of hOAT3 was activated de novo by forced expression of HNF1alpha alone or both HNF1alpha and HNF1beta together with the concomitant DNA demethylation in human embryonic kidney 293 cells that lack expression of endogenous HNF1alpha
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http://www.ncbi.nlm.nih.gov/pubmed/16793932
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Selventa
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2014-07-03T14:33:01.374+02:00
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