@prefix this: . @prefix rdfs: . @prefix xsd: . @prefix sio: . @prefix ncit: . @prefix lld: . @prefix miriam-gene: . @prefix miriam-pubmed: . @prefix eco: . @prefix wi: . @prefix prov: . @prefix pav: . @prefix prv: . @prefix dcterms: . @prefix np: . @prefix dgn-np: . @prefix dgn-gda: . @prefix dgn-void: . dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_head { this: np:hasAssertion dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_assertion; np:hasProvenance dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_provenance; np:hasPublicationInfo dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_publicationInfo; a np:Nanopublication . dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_assertion a np:Assertion . dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_provenance a np:Provenance . dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_publicationInfo a np:PublicationInfo . } dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_assertion { miriam-gene:948 a ncit:C16612 . lld:C0020538 a ncit:C7057 . dgn-gda:DGN7f4971b2d576deb976c70df58f55add0 sio:SIO_000628 miriam-gene:948, lld:C0020538; a sio:SIO_001121 . } dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_provenance { dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_assertion dcterms:description "[In the current studies, we have demonstrated (1) that a gene or genes responsible for a whole spectrum of cardiovascular risk factors mapped to a limited segment of the centromeric region of rat chromosome 4, (2) that a spontaneous deletion in the gene for Cd36 that encodes a fatty acid transporter and is located directly at the peak of QTL linkages on chromosome 4 has been indirectly linked to the transmission of insulin resistance, defective fatty acid metabolism, and increased blood pressure, and (3) based on complementation analysis in two transgenic lines expressing wild-type Cd36 on the genetic background of the SHR strain harboring the deletion variant of Cd36, we have established that defective Cd36 can be a determinant of disordered fatty acid metabolism, glucose intolerance, and insulin resistance in spontaneous hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en; wi:evidence dgn-void:source_evidence_literature; sio:SIO_000772 miriam-pubmed:11140856; prov:wasDerivedFrom dgn-void:befree-20150227; prov:wasGeneratedBy eco:ECO_0000203 . dgn-void:befree-20150227 pav:importedOn "2015-02-27"^^xsd:date . dgn-void:source_evidence_literature a eco:ECO_0000212; rdfs:comment "Gene-disease associations inferred from text-mining the literature."@en; rdfs:label "DisGeNET evidence - LITERATURE"@en . } dgn-np:NP293681.RAPxem7aUKmAiqZBIeG3vVhCNHrwp90nSyXCP2tuor1_g130_publicationInfo { this: dcterms:created "2015-08-25T14:40:28+02:00"^^xsd:dateTime; dcterms:rights ; dcterms:rightsHolder dgn-void:IBIGroup; dcterms:subject sio:SIO_000983; prv:usedData dgn-void:disgenetv3.0rdf; pav:authoredBy , , , , ; pav:createdBy ; pav:version "v3.0.0.0" . dgn-void:disgenetv3.0rdf pav:version "v3.0.0" . }