@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix rgd: . @prefix geneProductOf: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: rgd:2868; a Protein: . sub:_2 geneProductOf: rgd:3399; a Protein: . sub:_3 occursIn: species:10116; rdf:object sub:_2; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(RGD:Igf1) -> p(RGD:Prkcz)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "1.4" . sub:_4 prov:value "A stimulatory 15 mM glucose treatment increased the PKC zeta activity by 1.5 fold above that at zero glucose, and in the added presence of IGF1 the activity was further increased 10.4 fold. The addition of 0.4 mM palmitate/1% BSA enhanced IGF1induced PKC zeta activity in the presence of glucose by 15.3 fold above that at zero glucose, which was 47% higher than the level in equivalent INS1 cells incubated in the absence of FFA."; prov:wasQuotedFrom pubmed:11145586 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:11145586; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:29:53.851+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }