@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix hasPart: . @prefix ProteinComplex: . @prefix do: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 hasPart: hgnc:1582, sub:_2; occursIn: do:3068, species:9606; a ProteinComplex: . sub:_2 geneProductOf: hgnc:9956; a Protein: . sub:assertion rdfs:label "complex(p(HGNC:RELB),g(HGNC:CCND1))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value "(from text - refer to Figure 6) This analysis indicated significant binding (p < 0.05) of RelB to NFkB sites upstream of 7 genes in T98G cells maintained in serum, including known NFkB sites upstream of BIRC3/cIAP2, CCL2, CCND1, GABPB2, MAT2A (site II), PLAU and PTX3, and a novel site in MAT2A (site I)...RelB has a transactivation domain and the predicted sites occurred upstream of genes that were down-regulated in response to PI 3-kinase inhibition."; prov:wasQuotedFrom pubmed:18226221 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:18226221; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:16.147+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }