@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix go: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix hasAgent: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 hasAgent: sub:_2; a go:0003824 . sub:_2 geneProductOf: hgnc:2558; a Protein: . sub:_3 geneProductOf: hgnc:5344; a Protein: . sub:_4 occursIn: obo:EFO_0002795, species:9606; rdf:object sub:_3; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "cat(p(HGNC:CX3CR1)) -> p(HGNC:ICAM1)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_6; pav:version "20131211" . sub:_5 prov:value "We transfected CX3CR1-interfering RNA or control siRNA into cultured HUVECs and exposed these cells to s-FKN. HUVECs with CX3CR1 knockdown expressed less ICAM-1 mRNA and protein compared with cells transfected with control siRNA (Figure 4a and 4b), indicating that FKN does activate ICAM-1 in ECs through CX3CR1."; prov:wasQuotedFrom pubmed:17885215 . sub:_6 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:17885215; prov:wasDerivedFrom beldoc:, sub:_5 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:12.533+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }