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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
http://purl.bioontology.org/ontology/MSH/D017209
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a(CHEBI:lipopolysaccharide) -| (bp(GOBP:"response to endoplasmic reticulum stress") -> bp(MESHPP:Apoptosis))
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bp(GOBP:"response to endoplasmic reticulum stress") -> bp(MESHPP:Apoptosis)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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The induction of CHOP by LPS was delayed and weak, in comparison with CHOP induction by ER stress-inducer thapsigargin. In addition, LPS-pre-treatment or overexpression of ER chaperone, IgH chain binding protein (BiP), prevented ER stress-mediated apoptosis. LPS plus IFN-gamma-treated macrophages produce a larger amount of nitric oxide (NO) in comparison with LPS-treated cells. Treatment with the NO donor, SNAP (S-nitro-N-acetyl-dl-penicillamine), induces CHOP at an earlier period than LPS treatment. The depletion of NO retards CHOP induction and prevents apoptosis in LPS plus IFN-gamma-treated cells.
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http://www.w3.org/ns/prov#wasQuotedFrom
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Selventa
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2014-07-03T14:33:37.528+02:00
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