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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAN85sWn1A8fcJdiCvuxbYcvrAdm8SR1dP1nPF30C_ey8#_5 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAN85sWn1A8fcJdiCvuxbYcvrAdm8SR1dP1nPF30C_ey8#_4 http://www.w3.org/ns/prov#value The absence of PDE1C expression in all contractile/quiescent VSMC studied so far indicates that Ca2+ stimulated hydrolysis is unlikely to regulate cAMP levels in this VSMC phenotype. Thus, whereas PDE1C expression was markedly induced in VSMC cultured from several human arteries, no such induction was observed when rat, monkey, or bovine VSMC were similarly cultured (Rose et al., 1997; Liu and Maurice, 1998; Palmer and Maurice, 2000). 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