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[Additional mechanisms have been suggested for the ability of flavonoids to delay the initiation of and/or slow the progression of AD-like pathology and related neurodegenerative disorders, including a potential to inhibit neuronal apoptosis triggered by neurotoxic species (e.g., oxidative stress and neuroinflammation) or disrupt amyloid ? aggregation and effects on amyloid precursor protein processing through the inhibition of ?-secretase (BACE-1) and/or activation of ?-secretase (ADAM10).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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