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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAKyf6TrrH7kbzYAAA-NxptEwNCxBFxUH1nL5ZaMyfFN4#_5 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAKyf6TrrH7kbzYAAA-NxptEwNCxBFxUH1nL5ZaMyfFN4#_4 http://www.w3.org/ns/prov#value Initiation of these checkpoints is dependent on the transient recruitment of the MRE11/RAD50/NBS1 (MRN) complex at DSB sites, followed by the recruitment/activation of ataxia–telangiectasia mutated (ATM) a member of the family of phosphoinositide-3-kinase-related kinases (PIKKs) (Su, 2006). 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