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http://www.tkuhn.ch/bel2nanopub/RAJf7xNewjIdzg3nCibW5Tj4IlaIaF6-iwNZ_W2NPDapI#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RAJf7xNewjIdzg3nCibW5Tj4IlaIaF6-iwNZ_W2NPDapI#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0016301
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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kin(p(HGNC:ATM)) -> (p(HGNC:E2F1) -> bp(MESHPP:Apoptosis))
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http://www.w3.org/2000/01/rdf-schema#label
p(HGNC:E2F1) -> bp(MESHPP:Apoptosis)
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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http://www.w3.org/ns/prov#value
Moreover, induction of both apoptosis and p53 phosphorylation by E2F1 is abolished by caffeine; this is an inhibitor of the ATM and ATR kinases [33], supporting the notion that E2F1 uses the ATM signaling pathway to induce p53 and CHK2 phosphorylation and thereby apoptosis
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.tkuhn.ch/bel2nanopub/RAJf7xNewjIdzg3nCibW5Tj4IlaIaF6-iwNZ_W2NPDapI#assertion
http://www.w3.org/ns/prov#wasDerivedFrom
http://www.tkuhn.ch/bel2nanopub/RAJf7xNewjIdzg3nCibW5Tj4IlaIaF6-iwNZ_W2NPDapI#_6
http://www.tkuhn.ch/bel2nanopub/RAJf7xNewjIdzg3nCibW5Tj4IlaIaF6-iwNZ_W2NPDapI#pubinfo
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http://purl.org/dc/terms/created
2014-07-03T14:32:57.079+02:00
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