@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix chebi: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: obo:CLO_0001601, species:9606; rdf:object chebi:5586; rdf:predicate belv:decreases; rdf:subject chebi:5437; a rdf:Statement . sub:assertion rdfs:label "a(CHEBI:glutathione) -| a(CHEBI:\"hydrogen peroxide\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "20131211" . sub:_2 prov:value "H2O2 generated by either GO or cigarette smoke is almost completely ablated in the presence of GSH (Fig. 2C). Furthermore, there is a concomitant inhibition of GO- and cigarette smoke-induced EGFR phosphorylation in the presence of GSH, whereas activation by EGF is not affected (Fig. 2C)."; prov:wasQuotedFrom pubmed:17971399 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:17971399; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:13.478+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }