@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix chebi: . @prefix mesh: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: obo:CL_0000169, obo:UBERON_0002385, species:9606; rdf:object mesh:D007333; rdf:predicate belv:decreases; rdf:subject chebi:6801; a rdf:Statement . sub:assertion rdfs:label "a(CHEBI:metformin) -| bp(MESHPP:\"Insulin Resistance\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "20131211" . sub:_2 prov:value "The greater suppression of hepatic glucose production with glyburide (76) has been offered as an explanation for the higher incidence of hypoglycemia Metformin enhances the sensitivity of both hepatic and peripheral tissues (primarily muscle) to insulin (92-94) (Table 1, Figure 1). Metformin inhibits hepatic gluconeogenesis both in vitro (95) and in vivo"; prov:wasQuotedFrom pubmed:10454950 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:10454950; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dct:created "2014-07-03T14:31:31.657+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }