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http://www.tkuhn.ch/bel2nanopub/RAGEXrFEUB--146XaPwbGqfWricZB8swXutLcOmrQztZ0
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
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http://www.tkuhn.ch/bel2nanopub/RAGEXrFEUB--146XaPwbGqfWricZB8swXutLcOmrQztZ0#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11766
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
http://amigo.geneontology.org/amigo/term/GO:0048591
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http://www.w3.org/2000/01/rdf-schema#label
p(HGNC:TGFB1) -| bp(GOBP:"cell growth")
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
http://purl.org/dc/elements/1.1/description
Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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http://www.w3.org/ns/prov#value
Transforming growth factor-beta1 (TGF-beta1) can induce rapid growth arrest and apoptosis in hepatic cells. Its growth suppressive effects appear to be linked to decreased phosphorylation of the protein product of the retinoblastoma gene, pRb. Treatment of HuH-7 cells with TGF-beta1 inhibited expression and phosphorylation of pRb, and also induced apoptosis
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http://www.w3.org/ns/prov#wasQuotedFrom
http://www.ncbi.nlm.nih.gov/pubmed/8649852
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
http://www.ncbi.nlm.nih.gov/pubmed/8649852
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http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RAGEXrFEUB--146XaPwbGqfWricZB8swXutLcOmrQztZ0
http://purl.org/dc/terms/created
2014-07-03T14:33:52.133+02:00
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