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http://www.tkuhn.ch/bel2nanopub/RAGAjLEGM3wZL_Chq7tyKxgQjfH1Sa63XzDMET0M-wAJ0#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.informatics.jax.org/marker/MGI:94909
http://www.tkuhn.ch/bel2nanopub/RAGAjLEGM3wZL_Chq7tyKxgQjfH1Sa63XzDMET0M-wAJ0#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://resource.belframework.org/belframework/20131211/namespace/selventa-legacy-chemicals/Calcium
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http://amigo.geneontology.org/amigo/term/GO:0006950
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bp(GOBP:"response to stress") -> (p(MGI:Dmd) -| a(SCHEM:Calcium))
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http://www.w3.org/2000/01/rdf-schema#label
p(MGI:Dmd) -| a(SCHEM:Calcium)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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It is likely that chronic elevation of [Ca(2+)] in the cytosol in response to stress is an initiating event for apoptosis and/or necrosis in Duchenne muscular dystrophy or mdx muscle and that alterations in mitochondrial function and metabolism are involved. Other cellular signalling pathways (e.g. nitric oxide) might also be affected.
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Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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2014-07-03T14:31:53.462+02:00
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