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http://semanticscience.org/resource/SIO_000139
http://www.tkuhn.ch/bel2nanopub/RAFo-y_MpKwo5EwsjI1TSNrhxtO0KEcG9mDpNbTuptoho#_2
http://www.tkuhn.ch/bel2nanopub/RAFo-y_MpKwo5EwsjI1TSNrhxtO0KEcG9mDpNbTuptoho#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0003824
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=270
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=8768
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cat(p(HGNC:PARP1)) -> tloc(p(HGNC:AIFM1),GOCCID:0005739,GOCCID:0005737)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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http://www.w3.org/ns/prov#value
We found, based on genetic knockouts and pharmacological inhibition, that c-Jun N-terminal kinase (JNK), especially JNK1, but not the other groups of mitogen-activated protein kinase, is required for PARP-1-induced mitochondrial dysfunction, apoptosis-inducing factor translocation, and subsequent cell death.
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Selventa
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http://www.tkuhn.ch/bel2nanopub/RAFo-y_MpKwo5EwsjI1TSNrhxtO0KEcG9mDpNbTuptoho#assertion
http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RAFo-y_MpKwo5EwsjI1TSNrhxtO0KEcG9mDpNbTuptoho#pubinfo
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http://purl.org/dc/terms/created
2014-07-03T14:32:55.777+02:00
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