@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix go: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:11766; a Protein: . sub:_2 occursIn: obo:CL_0000451, species:9606; rdf:object go:0097028; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:TGFB1) -> bp(GOBP:\"dendritic cell differentiation\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value "In particular, DC differentiated in the presence of exogenously added TGF-b, IL-10, GM-CSF, and IL-4 possess the immunophenotypic and functional features of immature DC and induce CD4+CD25+ Treg cells, which suppress the response of antigen-primed CD4+ T cells to allogeneic, normal mature DC [136]. In addition, the release of proinflammatory cytokines, i.e., IL-1b, IL-6, TNF-a, and most markedly, IL-12, is abolished after IL-10 treatment [137]."; prov:wasQuotedFrom pubmed:17652446 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:17652446; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:12.010+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }