@prefix this: <http://www.tkuhn.ch/bel2nanopub/RAF1CUNHYhnwbGiNwCsfcHA3l9T3tdLliRMB_jLL7WW-A> .
@prefix sub: <http://www.tkuhn.ch/bel2nanopub/RAF1CUNHYhnwbGiNwCsfcHA3l9T3tdLliRMB_jLL7WW-A#> .
@prefix beldoc: <http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix rdf: <http://www.w3.org/1999/02/22-rdf-syntax-ns#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix dct: <http://purl.org/dc/terms/> .
@prefix dce: <http://purl.org/dc/elements/1.1/> .
@prefix pav: <http://purl.org/pav/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix belv: <http://www.selventa.com/vocabulary/> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix schem: <http://resource.belframework.org/belframework/20131211/namespace/selventa-legacy-chemicals/> .
@prefix obo: <http://purl.obolibrary.org/obo/> .
@prefix occursIn: <http://purl.obolibrary.org/obo/BFO_0000066> .
@prefix species: <http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=> .
@prefix pubmed: <http://www.ncbi.nlm.nih.gov/pubmed/> .
@prefix orcid: <http://orcid.org/> .

sub:Head {
  this: np:hasAssertion sub:assertion;
    np:hasProvenance sub:provenance;
    np:hasPublicationInfo sub:pubinfo;
    a np:Nanopublication .
}

sub:assertion {
  sub:_1 occursIn: obo:CL_0000233, species:9606;
    rdf:object schem:Calcium;
    rdf:predicate belv:increases;
    rdf:subject schem:Inositol%201%2C4%2C5-trisphosphate;
    a rdf:Statement .
  
  sub:assertion rdfs:label "a(SCHEM:\"Inositol 1,4,5-trisphosphate\") -> a(SCHEM:Calcium)" .
}

sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements.";
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
    dce:title "BEL Framework Large Corpus Document";
    pav:authoredBy sub:_3;
    pav:version "20131211" .
  
  sub:_2 prov:value "After the initial adhesion of platelets to the extracellular matrix, the repair process requires a rapid response to autocrine and paracrine mediators, including adenosine diphosphate (ADP), thrombin, epinephrine, and thromboxane A2. These mediators amplify and sustain the initial platelet response (Figure 1Figure 1Agonists, Receptors, and Effector Systems in Platelet Activation.), and they recruit circulating platelets from the flowing blood to form a growing hemostatic plug. Most agonists that activate platelets operate through G-protein?coupled receptors.14 The final pathway for all agonists is the activation of the platelet integrin glycoprotein IIb/IIIa (?IIb?3), the main receptor for adhesion and aggregation.15 ";
    prov:wasQuotedFrom pubmed:18077812 .
  
  sub:_3 rdfs:label "Selventa" .
  
  sub:assertion prov:hadPrimarySource pubmed:18077812;
    prov:wasDerivedFrom beldoc:, sub:_2 .
}

sub:pubinfo {
  this: dct:created "2014-07-03T14:33:14.110+02:00"^^xsd:dateTime;
    pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}