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http://www.tkuhn.ch/bel2nanopub/RADq4L0QM9pIOjEocOG8vyysrmhFpdB_S-wYDUHSdqjWE#_1
http://purl.obolibrary.org/obo/BFO_0000066
http://purl.obolibrary.org/obo/UBERON_0002370
http://www.tkuhn.ch/bel2nanopub/RADq4L0QM9pIOjEocOG8vyysrmhFpdB_S-wYDUHSdqjWE#_1
http://purl.obolibrary.org/obo/BFO_0000066
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
http://purl.bioontology.org/ontology/MSH/D006965
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http://www.w3.org/2000/01/rdf-schema#label
bp(GOBP:"cell proliferation") -> path(MESHD:Hyperplasia)
http://www.tkuhn.ch/bel2nanopub/RADq4L0QM9pIOjEocOG8vyysrmhFpdB_S-wYDUHSdqjWE#provenance
http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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http://www.w3.org/ns/prov#value
Targeted disruption of the murine p27(Kip1) gene caused a gene dose-dependent increase in animal size without other gross morphologic abnormalities. All tissues were enlarged and contained more cells, although endocrine abnormalities were not evident. Thymic hyperplasia was associated with increased T lymphocyte proliferation
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RADq4L0QM9pIOjEocOG8vyysrmhFpdB_S-wYDUHSdqjWE#pubinfo
http://www.tkuhn.ch/bel2nanopub/RADq4L0QM9pIOjEocOG8vyysrmhFpdB_S-wYDUHSdqjWE
http://purl.org/dc/terms/created
2014-07-03T14:33:52.116+02:00
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