@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix sdis: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix do: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:108420; a Protein: . sub:_2 occursIn: do:162, obo:CL_0000066, obo:UBERON_0002048, species:10090; rdf:object sdis:tissue%20damage; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(MGI:Nfe2l2) -| path(SDIS:\"tissue damage\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value "Relative to wild-type mice, lung hyperpermeability, inflammation, and epithelial cell injury were enhanced in Nrf2?/?mice (Cho et al., 2002a). Consistentwith a protective role for the Nrf2-ARE pathway during lung toxicity by hyperoxia,Nrf2–DNAbinding activity and expression of multiple antioxidant enzymes (e.g., GSTs, NQO1, UDP-glucuronyl transferase [UGT], GCLc, GCLm, HO-1, GPx2) were markedly suppressed in Nrf2?/? mice."; prov:wasQuotedFrom pubmed:19646463 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:19646463; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:36.025+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }