@prefix dc: . @prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix go: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:1349419; a Protein: . sub:_2 occursIn: species:10090; rdf:object go:0006979; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(MGI:Aifm1) -| bp(GO:\"response to oxidative stress\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "1.4" . sub:_3 prov:value "AIF is a mitochondrial oxidoreductase that translocates to the nucleus leading to nuclear condensation and apoptosis18. In addition to its role in inducing apoptosis, AIF has a potent redox function in vitro, which is separable from its apoptogenic activity20. Many oxidoreductases have been shown to have important roles in maintaining intracellular free radical homeostasis"; prov:wasQuotedFrom pubmed:12353028 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12353028; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dc:created "2014-07-03T14:30:06.187+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }