@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix mesh: . @prefix species: . @prefix occursIn: . @prefix obo: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:990; a Protein: . sub:_2 occursIn: obo:CL_0000084, obo:UBERON_0002370, species:9606; rdf:object mesh:D017209; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:BCL2) -| bp(MESHPP:Apoptosis)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value "These reported that overexpression of E2F1 suppresses the expression of two apoptotic antagonists, BCL-2 and its family member MCL-1 (myeloid leukaemia cell differentiation protein 1), thereby leading to apoptosis.In both cases, transcriptional repression is direct and dependent on the DNA-binding domain of E2F1."; prov:wasQuotedFrom pubmed:16530485 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:16530485; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:32:57.090+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }