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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RADHPHpLbTuSP6qUBb1w84RZFAnA8x_RRh4EUvGzxM56s#_5 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RADHPHpLbTuSP6qUBb1w84RZFAnA8x_RRh4EUvGzxM56s#_4 http://www.w3.org/ns/prov#value Attempts to extend these findings in vivo, however, were initially hampered by perinatal lethality of homozygote C/EBPa null mice, which perish due to hypoglycemia secondary to a failure of hepatic gluconeogenesis [13]. Two different groups subsequently developed ways to work around this problem. One group expressed C/EBPb from the endogenous C/EBPa locus (so-called C/EBPb/b mice) [14]. This rescued the hepatic phenotype, presumably because the requirement for a C/EBP in liver is not restricted to the C/EBPa isoform. 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