sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_5 ;
    pav:version "1.4" .
  sub:_4 prov:value "Attempts to extend these findings in vivo, however, were initially hampered by perinatal lethality of homozygote C/EBPa null mice, which perish due to hypoglycemia secondary to a failure of hepatic gluconeogenesis [13]. Two different groups subsequently developed ways to work around this problem. One group expressed C/EBPb from the endogenous C/EBPa locus (so-called C/EBPb/b mice) [14]. This rescued the hepatic phenotype, presumably because the requirement for a C/EBP in liver is not restricted to the C/EBPa isoform. In white fat, however, C/EBPa function could not be replaced by C/EBPb, and these animals had small fat pads with reduced lipid content per cell." ;
    prov:wasQuotedFrom pubmed:15936931 .
  sub:_5 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:15936931 ;
    prov:wasDerivedFrom beldoc: , sub:_4 .
}