@prefix dc: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix chebi: .
@prefix schem: .
@prefix species: .
@prefix occursIn: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 occursIn: species:9606;
rdf:object schem:Calcium;
rdf:predicate belv:increases;
rdf:subject chebi:26034;
a rdf:Statement .
sub:assertion rdfs:label "a(CHEBI:\"phosphatidylinositol 3-phosphate\") -> a(SCHEM:Calcium)" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_3;
pav:version "1.4" .
sub:_2 prov:value "Collectively, not only does Btk positively regulate PKCbI, but PKCb can negatively regulates Btk activity as a feedback loop inhibitor (Fig. 2). apparently through this mechanism, Btk positively regulates the membrane translocation and activity of PKCbI Second messengers generated by PLCg, i.e., DAG and IP3 (subsequent Ca 2+ increase), activate various PKC isoforms including the I isoform.";
prov:wasQuotedFrom pubmed:12417015 .
sub:_3 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:12417015;
prov:wasDerivedFrom beldoc:, sub:_2 .
}
sub:pubinfo {
this: dc:created "2014-07-03T14:30:07.064+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}