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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAD9v35FaD_5C8yYlOqDjRhx2799OKHYYO_vTKD57rQ-0#_5 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAD9v35FaD_5C8yYlOqDjRhx2799OKHYYO_vTKD57rQ-0#_4 http://www.w3.org/ns/prov#value Increased oxygen radical production may not only contribute to posttransplant ischemia-reperfusion injury but also to acute rejection of renal allografts. Xanthine oxidoreductase (XOR) may constitute a relevant reactive oxygen species (ROS) source. CONCLUSIONS: A major part of ROS generation in acute rejection was contributed by XOR. 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