@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix entrez: . @prefix proteinModification: . @prefix psimod: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix do: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:1784; a Protein: . sub:_2 belv:variantOf entrez:5424; a proteinModification:, psimod:00696 . sub:_3 occursIn: do:162, obo:CL_0000236, obo:UBERON_0000468, species:9606; rdf:object sub:_2; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:CDKN1A) -| p(EGID:5424,pmod(P,S))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "p53 also contributes to the G2/M checkpoint through its transactivation of p21 and 14-3-3. P21 effectively blocks the phosphorylation of CDC2, initiating the onset of the G2/M cell-cycle arrest. 14-3-3 sequesters CDC25C in the cytoplasm and promotes the activation ofWee1, a tyrosine kinase that negatively regulates CDC2, thus blocking entry into mitosis."; prov:wasQuotedFrom pubmed:18285820 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:18285820; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:17.496+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }