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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RACYObbkBRFJ5MR2hDJ8rdoGTbhozdxGM-n-79eQDFTRI#assertion
http://www.tkuhn.ch/bel2nanopub/RACYObbkBRFJ5MR2hDJ8rdoGTbhozdxGM-n-79eQDFTRI#_1
http://semanticscience.org/resource/SIO_000139
http://www.tkuhn.ch/bel2nanopub/RACYObbkBRFJ5MR2hDJ8rdoGTbhozdxGM-n-79eQDFTRI#_2
http://www.tkuhn.ch/bel2nanopub/RACYObbkBRFJ5MR2hDJ8rdoGTbhozdxGM-n-79eQDFTRI#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0003824
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11849
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http://resource.belframework.org/belframework/1.0/namespace/selventa-legacy-diseases/viral%20infection
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path(SDIS:"viral infection") -> cat(p(HGNC:TLR3))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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http://www.w3.org/ns/prov#value
Two IKK-related proteins - IKK-e (also known as inducible IKK,IKKi) and TBK1 (TRAF-family-memberassociated NF-kB activator (TANK)-binding kinase 1; also known as NF-kB-activating kinase,NAK) - have recently been identified as the kinases that phosphorylate IRF3 in response to viral infection and stimulation of TLR3
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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2014-07-03T14:30:25.542+02:00
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