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http://www.tkuhn.ch/bel2nanopub/RACM36cnCAzyf0iyjEmJWp3oxLj6FwwG5bBsSDXke61-0
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
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http://www.tkuhn.ch/bel2nanopub/RACM36cnCAzyf0iyjEmJWp3oxLj6FwwG5bBsSDXke61-0#_1
http://purl.obolibrary.org/obo/RO_0002204
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
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http://amigo.geneontology.org/amigo/term/GO:0001666
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http://www.w3.org/2000/01/rdf-schema#label
bp(GOBP:"response to hypoxia") -| r(HGNC:LPL)
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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BEL Framework Large Corpus Document
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http://www.w3.org/ns/prov#value
Both hypoxia-mimetic deferoxamine mesylate (DFO) and TGF-beta1 inhibited adipocyte differentiation (1.0% versus the control at 15 microm DFO and 1.4% versus the control at 1 ng/ml TGF-beta1) and adipocyte gene expression (peroxisome proliferator-activated receptor-gamma2 and lipoprotein lipase) in human MSCs after 21 days of treatment. Hypoxia (2% O(2)) and DFO (but not TGF-beta1) increased hypoxia-inducible factor-1alpha as shown by Western blotting.
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http://www.w3.org/ns/prov#wasQuotedFrom
http://www.ncbi.nlm.nih.gov/pubmed/15845540
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.tkuhn.ch/bel2nanopub/RACM36cnCAzyf0iyjEmJWp3oxLj6FwwG5bBsSDXke61-0#pubinfo
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http://purl.org/dc/terms/created
2014-07-03T14:32:43.497+02:00
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