@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:6407; a Protein: . sub:_2 geneProductOf: hgnc:1784; a Protein: . sub:_3 occursIn: obo:CLO_0001601, species:9606; rdf:object sub:_2; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:KRAS) -> p(HGNC:CDKN1A)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "A549 cells harbor the K-ras mutation. Active Ras can induce p21Cip1/Waf1 expression and cell cycle arrest via the p14Arf-p53 pathway, which is defective in A549 cells (Supplementary Fig. S1A) and via mitogen-activated protein kinase (MAPK) signaling (43)."; prov:wasQuotedFrom pubmed:19223555 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:19223555; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:32.254+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }