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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RABUmJVTzMFKUGI4b4pBSBG_C1LdfwG5lBP80muhupDAA#_5 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RABUmJVTzMFKUGI4b4pBSBG_C1LdfwG5lBP80muhupDAA#_4 http://www.w3.org/ns/prov#value TGF-1beta treatment significantly decreased miR-155 expression levels (Fig. 6A). Although TGF-1beta also up-regulated the expression of the hAT1R gene (Fig. 6B), the time course for the increased production of hAT1R mRNA did not correlate well with the time course for the enhanced synthesis of hAT1R protein levels since these values remained higher than expected at the 8 and 12 h time points (Fig. 6, C versus B). 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