@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix microRNA: . @prefix hgnc: . @prefix geneProductOf: . @prefix Protein: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:31495; a microRNA: . sub:_2 geneProductOf: hgnc:1784; a Protein: . sub:_3 occursIn: obo:CLO_0009095, species:9606; rdf:object sub:_2; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "m(HGNC:MIR106B) -| p(HGNC:CDKN1A)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "Intriguingly, we confirmed that miR-106b and miR-93 endogenously expressed in Snu-16 cells posttranscriptionally regulate p21. In fact, their inhibition by ASOs enhanced the expression of p21 protein (Figure 4A). Conversely, upregulation of miR- 106b and miR-93 achieved by either oligonucleotide transfection (Figure 4B) or lentiviral transduction (Figure 4C) repressed p21 protein expression without significant changes in p21 mRNA levels (Figure 4D)."; prov:wasQuotedFrom pubmed:18328430 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:18328430; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:18.537+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }