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[To examine whether L3MBTL functions as a classic TSG in human hematologic malignancies, we screened a panel of 17 myeloid leukemia cell lines and peripheral blood or bone marrow samples from 29 MDS and 13 MPD patients for mutations in the entire L3MBTL coding sequence, including intron/exon splice junctions.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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