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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAANdB8GEWHBZDsYPxeR4as9nEaseTzonb9WJExLCUFQE#_5 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAANdB8GEWHBZDsYPxeR4as9nEaseTzonb9WJExLCUFQE#_4 http://www.w3.org/ns/prov#value from full text - As expected, MKK6EE expression caused phosphorylation of endogenous p38MAPK (Figure 3A). Moreover, cells ceased proliferation several days later (Figure 3B). This growth arrest was accompanied by increased SA-bgal activity (Supplementary Figure S3A), decreased BrdU incorporation (Supplementary Figure S3B), and a senescent morphology (Supplementary Figure S3C). 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