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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAABQ8f-A38Vdq6IgeWJZ0aPNvsIuwGEz0uNsS2n3XnIM#_5 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAABQ8f-A38Vdq6IgeWJZ0aPNvsIuwGEz0uNsS2n3XnIM#_4 http://www.w3.org/ns/prov#value We next assessed whether selective agonists would activate lung fibroblast PPAR-g. IMR-90 cells were transfected with a PPAR-dependent reporter construct, pFATP-luc, or control vector. Neither Tro nor Cig activated cells transfected with the control vector (data not shown), whereas Tro and Cig activated transcription of the PPAR reporter construct in a dose-dependent manner, with higher levels of activation demonstrated with Tro (Fig. 2). 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