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[Although TSA treatment significantly repressed canonical WNT signaling in MCF7 cells, it did not influence endogenous TCF4/?-catenin mRNA levels and activated TCF4/?-catenin-driven transcription from the GLCE promoter, indicating GLCE as a novel target for TCF4/?-catenin complex in breast cancer cells.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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