@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix schem: . @prefix hgnc: . @prefix proteinModification: . @prefix psimod: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 belv:variantOf hgnc:3321; a proteinModification:, psimod:00696 . sub:_2 occursIn: species:9606; rdf:object sub:_1; rdf:predicate belv:increases; rdf:subject schem:trans10%2Ccis12-Octadecadienoate; a rdf:Statement . sub:assertion rdfs:label "a(SCHEM:\"trans10,cis12-Octadecadienoate\") -> p(HGNC:ELK1,pmod(P))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "1.4" . sub:_3 prov:value "Stromal vascular cultures containing newly differentiated human adipocytes were pretreated with a vehicle (dimethyl sulfoxide) for 1 hour, then with 1 microM BRL49653 (PPARgamma agonist) for 24hours and subsequently treated with 30microM trans-10, cis-12 conjugated linoleic acid for an additional 24hours. CLA treatment conferred the ability of ERK1/2 to phosphorylate its nuclear substrate ELK1 in vitro. Pretreatment with BRL49653 blocked the CLA mediated ability of ERK1/2 to phosphorylate ELK-1"; prov:wasQuotedFrom pubmed:15067015 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:15067015; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:22.285+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }